CORRIGENDUM to Molecular Crosstalk Between Non-SMN-Related and SMN-Related Spinal Muscular Atrophy

Šoltić D, Fuller HR. Molecular Crosstalk Between Non-SMN-Related and SMN-Related Spinal Muscular Atrophy. Neuroscience Insights. 2020;15:1-3. doi: 10.1177/2633105520914301

On page 2, second paragraph, line 5, the in-text citation 6 was incorrectly mentioned as 7 in the below paragraph. The correct paragraph should read as:

Studies of GARS, which was found to be another downstream target of UBA1 pathways, provided further insights into the fundamental molecular mechanisms driving pathology in SMN-dependent SMA tissues.⁶ Increased expression of GARS in spinal cords from a mouse model of severe SMN-dependent SMA was restricted to sensory neurons and increasing the expression of UBA1 was sufficient to restore GARS levels and correct sensory neuron defects in severe SMN-dependent SMA mice.⁶ UBA1/GARS pathway dysregulation is therefore likely to be responsible, at least in part, for disrupted sensory neuron fate and altered sensory-motor connectivity in these mice.⁶