KremerDAktasOHartungHP. The complex world of oligodendroglial differentiation inhibitors. Ann Neurol2011; 69: 602–618.
2.
KotterMStadelmannCHartungHP. Enhancing remyelination in disease – can we wrap it up?Brain2011; 134: 1882–1900.
3.
CostaCMartinez-SaezEGutierrez-FrancoA. Expression of semaphorin 3A, semaphorin 7A and their receptors in multiple sclerosis lesions. Mult Scler2015; 21: 1632–1643.
4.
PasterkampRJGigerRJ. Semaphorin function in neural plasticity and disease. Curr Opin Neurobiol2009; 19: 263–274.
5.
De WinterFOudegaMLankhorstAJ. Injury-induced class 3 semaphorin expression in the rat spinal cord. Exp Neurol2002; 175: 61–75.
6.
PiatonGAigrotMSWilliamsA. Class 3 semaphorins influence oligodendrocyte precursor recruitment and remyelination in adult central nervous system. Brain2011; 134: 1156–1167.
7.
MajedHHChandranSNiclouSP. A novel role for Sema3A in neuroprotection from injury mediated by activated microglia. J Neurosci2006; 26: 1730–1738.
8.
TakamatsuHTakegaharaNNakagawaY. Semaphorins guide the entry of dendritic cells into the lymphatics by activating myosin II. Nat Immunol2010; 11: 594–600.
9.
KoppMABrommerBGatzemeierN. Spinal cord injury induces differential expression of the profibrotic semaphorin 7A in the developing and mature glial scar. Glia2010; 58: 1748–1756.
10.
HolmesSDownsAMFosberryA. Sema7A is a potent monocyte stimulator. Scand J Immunol2002; 56: 270–275.
11.
CzopikAKBynoeMSPalmN. Semaphorin 7A is a negative regulator of T cell responses. Immunity2006; 24: 591–600.
BoydAZhangHWilliamsA. Insufficient OPC migration into demyelinated lesions is a cause of poor remyelination in MS and mouse models. Acta Neuropathol2013; 125: 841–859.
14.
WilliamsAPiatonGAigrotMS. Semaphorin 3A and 3F: key players in myelin repair in multiple sclerosis?Brain2007; 130: 2554–2565.
15.
CantoETintoreMVillarLM. Validation of semaphorin 7A and ala-beta-his-dipeptidase as biomarkers associated with the conversion from clinically isolated syndrome to multiple sclerosis. J Neuroinflamm2014; 11: 181.