Abstract
Summary
Chronic neurogenic hypertension in dogs induced by moderator nerve section producing an increase in the central excitatory state of the medullary cardiovascular centers, is not permanently influenced to a significant degree by lesions of the hypothalamus or its descending pathways compatible with survival of the animals. It is suggested that the most important neurogenic homeostatic influence upon the arterial tension is exerted by the moderator nerves at the medullary level and that higher levels of the central nervous system act as temporary modulating influences in response to momentary needs rather than as chronic adjusters of the arterial tension.
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