Abstract
These experiments were performed with the object of ascertaining whether or not the contention of Blum as well as of Eppinger, Falta and Rudinger, that adrenalin stimulates the conversion of fat into dextrose, is well founded. On careful analysis of their data, one may find every reason to believe that the animals used for their adrenalin experiments were not glycogen free, and that the extra sugar eliminated after the administration of adrenalin did not come from the ingested fat, but from glycogen or from the sugar of the blood.
If a phlorhizinized animal be exposed to cold and rendered glycogen free, any intraperitoneal injection of adrenalin ought to be followed by an extra elimination of sugar and a rise in the D : N ratio, provided the theory of the conversion of fat into carbohydrate is true. That this is not the case will be seen from the accompanying protocols.
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