Abstract
Summary
The rate of oxygen uptake of heart muscle slices from rats in the terminal stages of prolonged severe hemorrhagic shock is significantly less than normal in the absence of substrate. In the presence of pyruvate (0.02 M) the rate of oxygen uptake is increased, but is still less than the normal rate. There is, therefore, no conclusive evidence of impaired ability of the heart muscle to oxidize pyruvate in these circumstances.
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