Abstract
Summary
We conclude that the increase in isovolumetrically developed intraventric-ular pressure is associated with an enhanced activity of cardiac sympathetic afferents traversing the left upper thoracic white rami communicantes, as reported by Hess et al. (8). Moreover, the sympathetic afferent nerve fibers respond to alteration in myocardial muscle length occurring in isobaric diastole and thus respond to a displacement stimulus as well as to a pressure stimulus.
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