Abstract
Summary
Neutrophilia, produced by bleeding or by parasitic infection, did not result in a larger than normal number of cells migrating into endotoxin-induced peritoneal exudates. These results fail to support but do not disprove the concept that neutro-phil loss from the blood may be controlled primarily by changes in the fractional turnover rate rather than by blood pool size. They indicate that the failure of exudate neutro-phils to increase in proportion to blood neu-trophils in patients with chronic myeloid leukemia may not be an abnormality as interpreted previously.
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