Abstract
Summary
The isolated, intralobar renal artery appears to be a useful preparation to study the actions of agents upon renal vasculature. It may be concluded that the interlobar arteries contribute to the renal vasoconstriction produce by norepinephrine and dopamine, but do not contribute to the renal vasodilatation produced by furosemide or small doses of dopamine. Hydrochlorothiazide may interfere with adrenergic vasoconstriction to an extent noted for other vascular beds. It is equivocal whether the larger renal arteries contribute to the renal vasoconstiction produced by thiazides.
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