Abstract
Summary
CAI produced by rabbits challenged with M. pneumoniae (MP), Streptococcus MG (MG) or L. monocytogenes (LM) were inactivated by absorption with those microorganisms or a lipopolysaccharide (LP) fraction thereof. CAI from sera of patients with MP infections were not inactivated by the intact microorganism, but by the LP fraction of MP. CAI from patients with malignant lymphoproliferative disorders was not absorbed by either. An antigenic component of MP is responsible for CAI in MP infections.
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