Abstract
Summary
It would seem that both phentolamine blockade and denervation affected the series-coupled circuits by lowering precapillary resistance and opening precapillary sphincters, thus making a larger capillary surface area available. Capillary hydrostatic pressure probably was also increased. The parallel-coupled circuits were affected by making A-V shunts available to elevated flow. These shunts are apparently in the skin and one could, therefore, expect blood flow to pass through the skin at an increasingly greater percentage of the total limb flow as the flow rate is increased.
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