Abstract
Summary
The net accumulation of (14C) amino acids in rat renal-cortical slices in vitro is inhibited by decreased concentrations of calcium or inorganic phosphate in the incubation media. Renal-cortical slices from para-thyroidectomized rats also were unable to accumulate amino acids normally. Vitamin D deficient rickets in rats was not associated with a defect in the ability of the kidney to transport (14C) amino acids. The role of Ca2+ and Pi in the maintenance of normal cellular function may contribute to the normal renal transport mechanism for amino acids.
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