Abstract
The role of nitric oxide (NO) signaling in the retina can be simply termed as "extensive." The picture remains incomplete, but it is now known that NO has many sites of production and action in the retina, both physiological and pathophysiological in nature. Perspectives from retinal neurophysiology and clinical pathology have merged in a number of studies examining NO action, but renewed emphasis is needed to discover the links between the roles of NO in the neurons, glia, and vasculature of the retina. NEUROSCIENTIST 3:357-360, 1997
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