BallantyneJ. W. (1902). In Manual of antenatal pathology & hygiene—The Foetus, p. 262. Edinburgh: Green.
2.
BairdD. (1962). Combined Textbook of Obstetrics and Gynaecology, 7th Ed., p. 91. Edinburgh: Livingstone.
3.
ten BergJ. A. G.GrotepassW. (1941). Onserzoek Waar Verschijnselen van Loodvergifliging als Gevolg van Loodhoudend Drinkwater.Utrecht: Dekker & van de Vegt.
4.
DieckmannW.WegnerC. R. (1934.) Studies of the blood in normal pregnancy. Arch, intern. Med., 53, p. 527.
5.
DonathW. F. (1955). T. soc. Geneesk., 33, 91.
6.
DonathW. F. (1956). Simple portable apparatus for the semi-quantitative determination of the coproporphyrin content in urine. Arh. Hig. Rada, 7, 77.
7.
GoldbergA.SmithJ. A.LochheadA. C. (1963). Treatment of lead-poisoning with oral penicillamine. Brit. med. J., 1, 1270.
8.
GoldblattM. W.GoldblattJ. (1956). Industrial medicine and hygiene, Vol. 3, p. 456 (ed. MereweatherE. R. A.). London: Butterworth.
9.
Haeger-AronsenB. (1960). Studies on urinary excretion of δ-aminolaerulic acid and other haem precursors in lead workers and lead-intoxicated rabbits. Scand. J. clin., Lab. Invest., 12, Suppl. 47, 21.
10.
HsiaD. Y. (1960) Inborn errors of metabolism, p. 19. Chicago: Year Book Publishers.
11.
IrvingJ. T. (1957). Calcium metabolism, p. 62. London: Methuen.
12.
KehoeR. A. (1961). The metabolism of lead in man in health and disease. J. roy. Inst. publ. Hlth,.24, 81.
13.
LaneR. E. (1949). The care of the lead worker. Brit. J. industr. Med., 6, 125.
14.
LaneR. E. (1964). Health control in inorganic lead industries. A follow-up of exposed workers. Arch. environm. Hlth., 8, 243.
15.
de LangenC. D.ten BergJ. A. G. (1948). Porphyrin in the urine as a first symptom of lead poisoning. Acta med. scand., 130, 37.
16.
MoncrieffA. A.KoumidesO. P.ClaytonB. E.PatrickA. D.RenwickA. G. C.RobertsG. E. (1964). Lead poisoning in children. Arch. Dis Childh., 39, 1.
17.
NishimuraH. (1964). Chemistry and Prevention of congenital anomalies, p. 64. Springfield, Ill., U.S.A.: Thomas.
18.
PorrittN. (1934). The menace and geography of eclampsia in England and Wales.Oxford: Oxford Univ. Press.
19.
StanburyJ. B.WyngaardenJ. B.FredericksonD. S. (1960). The metabolic basis of inherited disease, p. 438. McGraw-Hill.
20.
StewartC. P.StolmanA. (1960). Toxicology, Vol. I, p. 215. New York and London: Academic Press.
21.
TompsettS. L.AndersonA. B. (1935). Lead content of human tissues and excreta. Biochem. J., 29, 1851.
22.
WaardenbergP. J.FranceschettiA.KleinD. (1961). Genetics and ophthalmology, p. 730. Oxford: Blackwell.
23.
WaldmanR. K.SeidemanR. M. (1950). Reliability of the urinary porphyrin test for lead absorption. Arch, industr. hyg., 1, 290.
24.
WilsonA. T. (1964). Methods of prospecting for lead effects in general practice. Proc. roy. Soc. Med., 57, 257.
25.
ZielhuisR. L. (1961). Coproporphyrinuria in groups of workers as an index of inorganic lead absorption. Brit. J. industr. Med., 18, 58.