O'BryanT. You Can Fix Your Brain. New York: Rodale Books, 2018.
2.
HadjivassiliouM, GrünewaldRA, LawdenM, et al.Headache and CNS white matter abnormalities associated with gluten sensitivity. Neurology, 2001; 56:385–388.
3.
SnirO, MesinL, GidoniM, et al.Analysis of celiac disease autoreactive gut plasma cells and their corresponding memory compartment in peripheral blood using high-throughput sequencing. J Immunol, 2015; 194:5703–5712.
4.
HadjivassiliouM, GrünewaldRA, Davies-JonesGA. Gluten sensitivity as a neurological illness. J Neurol Neurosurg Psychiatry, 2002; 72:560–563.
5.
GabrielliM, CremoniniF, FioreG, et al.Association between migraine and Celiac disease: Results from a preliminary case-control and therapeutic study. Am J Gastroenterol, 2003; 98:625–629.
6.
CervioE, VoltaU, VerriM, et al.Sera from patients with celiac disease and neurologic disorders evoke a mitochondrial-dependent apoptosis in vitro. Gastroenterology, 2007; 133:195–206.
7.
YuXB, UhdeM, GreenPH, et al.Autoantibodies in the extraintestinal manifestations of celiac disease. Nutrients, 2018; 10:1123.
8.
AlaediniA, OkamotoH, BrianiC, et al.Immune cross-reactivity in celiac disease: Anti-gliadin antibodies bind to neuronal synapsin I. J Immunol, 2007; 178:6590–6595.
9.
PratesiR, GandolfiL, FriedmanH, et al.Serum IgA antibodies from patients with coeliac disease react strongly with human brain blood-vessel structures. Scand J Gastroenterol, 1998; 33:817–821.
10.
CaioG, VoltaU, SaponeA, et al.Celiac disease: A comprehensive current review. BMC Med, 2019; 17:142.
11.
Parra-MedinaR, Molano-GonzalezN, Rojas-VillarragaA, et al.Prevalence of celiac disease in Latin America: A systematic review and meta-regression. PLoS One, 2015; 10:e0124040.
12.
AlmeidaLM, CastroLC, UenishiRH, et al.Decreased prevalence of celiac disease among Brazilian elderly. World J Gastroenterol, 2013; 19:1930–1935.
13.
AzizI, LewisNR, HadjivassiliouM, et al.A UK study assessing the population prevalence of self-reported gluten sensitivity and referral characteristics to secondary care. Eur J Gastroenterol Hepatol, 2014; 26:33–39.
14.
SchuppanD, ZevallosV. Wheat amylase trypsin inhibitors as nutritional activators of innate immunity. Dig Dis, 2015; 33:260–263.
15.
VenturaA, MagazzùG, GrecoL. SIGEP Study Group for Autoimmune Disorders in Celiac Disease. Duration of exposure to gluten and risk for autoimmune disorders in patients with celiac disease. Gastroenterology, 1999; 117:297–303.
16.
ViljamaaM, KaukinenK, HuhtalaH, et al.Coeliac disease, autoimmune diseases and gluten exposure. Scand J Gastroenterol, 2005; 40:437–443.
17.
CarroccioA, D'AlcamoA, CavataioF, et al.High proportions of people with nonceliac wheat sensitivity have autoimmune disease or antinuclear antibodies. Gastroenterology, 2015; 149:596–603.
18.
LosurdoG, PrincipiM, IannoneA, et al.Extra-intestinal manifestations of non-celiac gluten sensitivity: An expanding paradigm. World J Gastroenterol, 2018; 24:1521–1530.
19.
MahlerM, FritzlerMJ. The dense fine speckled immunofluorescence pattern on HEp-2 cells for the diagnosis of systemic autoimmune diseases. Clin Dev Immunol, 2012; 2012:494356.
20.
PérezD, GilburdB, Cabrera-Marante Ó, et al. Predictive autoimmunity using autoantibodies: Screening for anti-nuclear antibodies. Clin Chem Lab Med, 2018; 56:1771–1777.
21.
WalkerMM, MurrayJA, RonkainenJ, et al.Detection of celiac disease and lymphocytic enteropathy by parallel serology and histopathology in a population-based study. Gastroenterology, 2010; 139:11211–11219.
22.
AlmeidaLM, GandolfiL, PratesiR, et al.Presence of DQ2.2 associated with DQ2.5 increases the risk for celiac disease. Autoimmune Diseases, 2016; 2016:5409653.
23.
CoburnJA, Vande VoortJL, LahrBD, et al.Human leukocyte antigen genetics and clinical features of self-treated patients on a gluten-free diet. J Clin Gastroenterol, 2013; 47:828–833.
24.
LeonardMM, SaponeA, CatassiC, et al.Celiac disease and nonceliac gluten sensitivity: A review. JAMA, 2017; 318:647–656.
25.
HollonJ, PuppaEL, GreenwaldB, et al.Effect of gliadin on permeability of intestinal biopsy explants from celiac disease patients and patients with non-celiac gluten sensitivity. Nutrients, 2015; 7:1565–1576.
26.
DragoS, El AsmarR, Di PierroM, et al.Gliadin, zonulin and gut permeability: Effects on celiac and non-celiac intestinal mucosa and intestinal cell lines. Scand J Gastroenterol, 2006; 41:408–419.
27.
Fritscher-RavensA, SchuppanD, EllrichmannM, et al.Confocal endomicroscopy shows food-associated changes in the intestinal mucosa of patients with irritable bowel syndrome. Gastroenterology, 2014; 147:1012–1020.
28.
VojdaniA, KharrazianD, MukherjeePS. The prevalence of antibodies against wheat and milk proteins in blood donors and their contribution to neuroimmune reactivities. Nutrients, 2013; 6:15–36.
29.
BoscoloS, LorenzonA, SblatteroD, et al.Anti transglutaminase antibodies cause ataxia in mice. PLoS One, 2010; 5:e9698.
30.
PruimboomL, de PunderK. The opioid effects of gluten exorphins: Asymptomatic celiac disease. J Health Popul Nutr, 2015; 33:24.
31.
SildorfSM, FredheimS, SvenssonJ, et al.Remission without insulin therapy on gluten-free diet in a 6-year old boy with type 1 diabetes mellitus. BMJ Case Rep, 2012; 2012:bcr0220125878.
TuntipopipatS, MuangnoiC, ChingsuwanroteP, et al.Anti-inflammatory activities of red curry paste extract on lipopolysaccharide-activated murine macrophage cell line. Nutrition, 2011; 27:479–487.
34.
LuQY, SummanenPH, LeeRP, et al.Prebiotic potential and chemical composition of seven culinary spice extracts. J Food Sci, 2017; 82:1807–1813.
35.
MinichDM. A review of the science of colorful, plant-based food and practical strategies for “Eating the Rainbow.”. J Nutr Metabo, 2019; 2019:2125070.
36.
LernerA, MatthiasT. Changes in intestinal tight junction permeability associated with industrial food additives explain the rising incidence of autoimmune disease. Autoimmun Rev, 2015; 14:479–489.
37.
LernerA, MatthiasT. Don't forget the exogenous microbial transglutaminases: It is immunogenic and potentially pathogenic. AIMS Biophys, 2016; 3:546–552.
38.
LernerA, MatthiasT. Transglutaminases in dysbiosis as potential environmental drivers of autoimmunity. Front Microbiol, 2017; 8:66.
39.
MatthiasT, JeremiasP, NeidhöferS, et al.The industrial food additive microbial transglutaminase, mimics the tissue transglutaminase and is immunogenic in celiac disease patients. Autoimmun Rev, 2016; 15:1111–1119.
40.
ZaniniB, MarulloM, VillanacciV, et al.Persistent intraepithelial lymphocytosis in celiac patients adhering to gluten-free diet is not abolished despite a gluten contamination elimination diet. Nutrients, 2016; 8:525.
41.
LudvigssonJF, MontgomerySM, EkbomA, et al.Small-intestinal histopathology and mortality risk in celiac disease. JAMA, 2009; 302:1171–1178.
42.
O'BryanT. The Autoimmune Fix. New York: Rodale Books, 2016.