Abstract
Summary
Low doses of l-triiodothyronine suppressed the mild forms of aminoacetonitrile poisoning in rats, whereas extremely high doses of thyroxine were necessary to accomplish the same result. On the basis of the prevention of aminonitrile lesions in rats, l-triiodothyronine was estimated to be over 100 times more active than l-thyroxine when administered orally and over 50 times when administered parenterally.
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