Abstract
Conclusions
1. Total circulatory occlusion alone does not slow the rate of the normother-mic or hypothermic heart. 2. Intracoronary acetylcholine will profoundly decrease the cardiac rate in the occluded heart. The duration of this effect is short at normal body temperatures but quite prolonged during hypothermia (24–27°C). 3. Intracoronary atropine readily reverses the acetylcholine effect on the heart. 4. Cardiac slowing or arrest affords an added protection to the hypothermic heart during total circulatory occlusion.
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