Abstract
Summary
1. Extracts of irradiated methyl linolenate and methyl linoleate inhibited respiratory and glycolytic activities of Ehrlich ascites tumor cells. Respiration showed a greater sensitivity than glycolysis to the oxidation products of both esters. 2. The oxidation products of methyl linolenate inhibited both glycolysis and respiration at lower concentrations than those for methyl linoleate. 3. In contrast to the normal tissues of the mouse, the oxidation of tumor cell lipids by aerobic incubation and ultraviolet light could not be demonstrated fatty acids of ascites tumor cells are presented.
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