Abstract
Summary
Fasting rats given large doses of sodium salicylate fail to deposit liver glycogen even when they are given a major stimulus for such deposition in the form of Compound F. Yet such animals continue to break down body protein at an accelerated rate as judged by the nitrogen balances. The level of muscle glycogen salicylate-treated rats is reduced and this effect is countered by concurrent treatment with Compound F. The significance of these findings is briefly discussed.
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