Abstract
Conclusions
1. The marked eosinophilia due to an experimental Trichina infection was greatly depressed but not abolished when ACTH was administered for short intervals. 2. The effects of ACTH in animals with artificially induced trichinosis were exhibited by (a) alterations of the inflammatory response to encysted larvae, (b) alterations in the bone marrow, and (c) the changes in the level of circulating eosinophils. 3. Although modification of the host response to Trichina infestation was obtained with ACTH there was no objective evidence that the infected rats benefited as a result of this treatment.
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