Abstract
Summary
On administration of desiccated thyroid in the diet, the syndrome of vit. B6 deprivation in the rat was not markedly altered. One apparent effect of thyroid-feeding was to eliminate the customary elevation in fasting blood urea consequent to vit. B6 restriction. This and an earlier study suggest that the biochemical defects of vit. B6 deprivation in the rat are not caused or prevented by alterations in thyroid activity.
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