Abstract
Summary and Conclusions
The pulmonary and brachial artery pressures and their responses to pressor and depressor agents have been studied in normotensive and hypertensive subjects. Pulmonary artery pressure is within the same range in both groups. The response of pulmonary artery pressure to pressor and depressor agents used is also the same in both groups. It is emphasized that the elevation of pulmonary artery pressure due to angiotonin, epinephrine, and nor-epinephrine is the same in hypertensive and normotensive subjects. It is concluded that none of these or any similarly acting substances plays a part in sustaining the blood pressure elevation in patients with chronic essential hypertension.
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