Abstract
Summary
1. Brief cortical vasoconstriction of the kidney followed occlusion of a renal artery not previously manipulated. When occlusion of the artery was maintained only a few seconds, an equally brief period of renal hyperemia followed. 2. More prolonged occlusion produced a short period of renal vasodilatation and congestion followed release of the occlusion. During this period of dilatation, the vessels did not respond by vasoconstriction to repeated occlusion of the renal artery.
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