Abstract
Summary
1. It has been observed that a pyridoxine deficiency in the rat does not disturb the normal conversion of tryptophan to liver pyridine nucleotides to any demonstrable extent. 2. Niacin fed to pyridoxine-deficient rats spares liver pyridine nucleotide concentrations. 3. Tryptophan fed in excess along with normal dietary protein in a complete ration increases liver pyridine nucleotides concentrations above normal demonstrating that the excess metabolite can be utilized rather than simply degraded and excreted.
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