Abstract
Summary
The carcinogen, 2-acetylaminofluorene, produced a riboflavin deficiency in rats, reducing the liver riboflavin below control values. Increasing the riboflavin content of the diet prevented the appearance of the deficiency, keeping the liver riboflavin at normal levels. In the absence of protein in the diet, however, the increased riboflavin did not prevent the development of the deficiency.
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