Abstract
Summary
In agreement with previous in vivo studies, liver homogenate metabolized acetaldehyde more rapidly than other tissues and without the formation of much acetoin. Methylene blue stimulated the aerobic metabolism of acetaldehyde in rat liver but not dog liver.
Skeletal muscle formed more acetoin from acetaldehyde than did brain or kidney. Pyruvate, α-ketoglutarate and phosphoglycerate increased the acetoin formation from acetaldehyde by muscle; pyruvate, a-ketoglutarate and glucose were effective stimulators of acetoin formation in brain.
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