Abstract
Summary
Tetraethylammonium chloride given intravenously to 9 normotensive patients in congestive failure caused a precipitous fall in venous pressure and a significant decrease in arterial pressure in every case. Compensated cardiacs and non-cardiac normotensive controls showed, no fall in venous pressure and a much smaller change in arterial pressure. Following digitalization, 2 cardiac subjects in failure lost some of their initial responsiveness to TEAC.
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