Abstract
Abstract
The hypothesis was tested that the hormone prolactin, a noncataractogenic agent, can influence cataractogenesis in rats fed on a D-galactose-rich diet. This hormone accelerated the cataractogenic process in females but was without significant effect in males. It was also found that the galactose diet itself induced cataract formation more rapidly in females than in males. Thus, prolactin may play a hitherto unsuspected role in acceleration of cataractogenesis, and this role is apparently influenced by the sex of the individual.
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