Abstract
Summary
Hypozincemia and hepatic sequestration of zinc occur in rats receiving a nonlethal scald injury as well as in rats with a lethal infection superimposed on the burn injury, somewhat in proportion to the severity of the injury. The amount of zinc accumulated by the liver is far in excess of that lost from serum and may be related to altered nitrogen distribution. Unlike liver, kidneys do not sequester zinc. The availability of food mutes, but does not obliterate either, the serum zinc depression or the hepatic accumulation of zinc.
Zinc redistribution during thermal injury with or without the complication of infection appears not to be related to alterations in serum albumin concentration.
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