Abstract
Summary
Melatonin was administered to male hamsters via subcutaneous Silastic or beeswax capsules. Treatments adequate to prevent gonadal regression in LD 2:22 or LD 6:18 also prevented such photoperiods from inducing neuroendocrine refractoriness to the regressive effects of short days. The results are discussed with attention to the mechanisms of melatonin's progonadal effects and the relationship of testicular regression to subsequent gonadal refractoriness.
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