Abstract
Summary
Atrial tamponade was used as a method to reduce atrial transmural pressure in dogs whose afferent pathways from cardiac receptors were interrupted by sympathectomy and vagotomy. Under these conditions, atrial tamponade caused decreases in renal sodium excretion and urine flow which were comparable to those previously observed in intact dogs. These observations lead us to conclude that the changes in renal function which occur during atrial tamponade are not caused by a reflex initiated by atrial receptors. Rather, the alterations in renal function appear to be attributable to unidentified changes in systemic hemodynamics that are elicited by atrial tamponade.
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