Abstract
Summary
The development of resistance to extrinsic fusion by Sendai virus in cells infected with myxoviruses or paramyxoviruses can be attributed to the destruction of sialo-glycoprotein receptors by newly synthesized viral neuraminidase incorporated in the cell membrane and inaccessible to antibody. Infection with any of the viruses which lack neuraminidase does not influence susceptibility to extrinsic fusion by Sendai virus, even though the cell membrane may undergo specific chemical and morphological alterations during viral maturation and release.
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