Abstract
Summary
The immediate cardiopulmonary effects following acute induced coronary occlusion are hyperpnea, systemic hypotension, and hyperventilation. The onset of hyperpnea and systemic hypotension is quite rapid. Arterial blood gas tensions reveal arterial hyperoxemia and hypocapnia, which may be partly due to hyperventilation. Arterial hypoxemia, following myocardial infarction, is a later event and possibly related to the pulmonary complications following myocardial infarction.
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