Abstract
Summary
Monkey liver has been used as a model system to study adaptive lipogenesis in primates. Fasting and refeeding produced marked fluctuation in overall fatty acid synthesis and related lipogenic enzyme activity. Adaptive enzyme formation does not appear to play a regulatory role in lipogenesis from monkey adipose tissue. Rates of incorporation of citrate-1,5-14C relative to other substrates appears to be sufficiently high in both monkey liver and adipose tissue to account for the citrate pathway in fatty acid biosynthesis.
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