Abstract
Summary
Boron hydrides inhibit AAT in the liver, heart, brain, and kidneys of normal rats. Decaborane does not cause a significant loss in activity in the tissues of pyridoxal-deficient rats. The AAT activity, when assayed with sufficient exogenous pyridoxal, is greater in the tissues from the pyridoxal-deficient animals than in the tissues from the normal animals. LDH, an enzyme that does not require pyridoxal, is moderately inhibited in the heart and kidney of both normal and pyridoxal-deficient animals, and in the brain of normal animals.
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