Abstract
Summary
In studies made of its general behavior in laboratory animals MVM was found to induce lethal infections in suckling rats, and runting accompanied by proliferations of virus to high titers in many organs in mice. MVM attacked the cerebellum of infant mice, but without sufficient damage to induce clinical ataxia. When H-l virus was studied in a comparable manner, however, it was found to induce marked ataxia accompanied by an extensive cerebellar hypoplasia. A survey of wild rats revealed that Hi antibodies to MVM (69.5%) were more prevalent than those to either H-l (61%) or to rat virus (34%).
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