Abstract
Summary
Autoregulation evoked by alterations in renal blood flow or renal arterial pressure to the in situ collateralized kidney was associated with only small changes in deep renal venous pressure and renal hilar lymphatic vessel pressure. Resistance calculations suggest that the change in resistance during renal autoregulation occurs predominantly in vessels upstream to the arcuate or intralobular veins. Thus, the present study fails to provide support for the “tissue pressure” hypothesis of renal autoregulation in the intact collateralized kidney.
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