Abstract
Summary
Respiration in the perfused hearts of vitamin E-deficient and -sufficient rats was studied and compared in the control, oligomycin, and cyanide treated hearts. No effect of vitamin E deficiency was noted in the total qO2, phosphorylated respiration or nonmitochondrial oxygen fixation. Studies of lipid peroxidation, as tested by the thiobarbituric acid (TBA) method, demonstrated high in vitro lipid peroxidation in the homogenates of hearts and livers of the vitamin E-deficient rats compared to the control. The TBA test in the heart perfusate of the two groups was the same.
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