Abstract
Summary
In studies in man, an intravenous infusion of 0.025 μg/kg/min of angiotensin for 5 hours significantly elevated the diastolic blood pressure, but had no effect on the urinary catecholamine or 3-methoxy-4-hydroxy mandelic acid (V.M.A.) excretion rates. The same dose of angiotensin infused intravenously for 1 hour had no effect on fasting blood glucose levels and did not cause glycosuria. It is concluded that catecholamine release does not play a significant role in the vascular responses observed with this dose of intravenous angiotensin in man.
Get full access to this article
View all access options for this article.