Abstract
Summary
Actinomycin D was administered at varying times after the administration of aldosterone to rats in order to determine if this inhibitor would either enhance or prolong the antinatriuretic action of aldosterone. No such effects were found, thus it would appear that an actinomycin D sensitive “represser” is not involved in terminating the sodium retaining action of the adrenocortical steroid hormone, aldosterone.
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