Abstract
Summary
The hyperglycemia produced by SFA can be significantly reduced by small amounts of insulin. The very high levels of plasma corticosterone found to occur following treatment with SFA can be transiently increased by the administration of insulin. These high levels are thought to be due to a SFA induced decrease in the hepatic inacti-vation of the adrenal hormone.
The authors wish to acknowledge the excellent technical assistance of Mrs. Alice C. Weil.
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