Abstract
Summary
Studies of erythrocyte survival using Cr51 and DFP32 labeled red cells in rats following end-to-side portacaval shunts disclosed no change in erythrocyte longevity. It is suggested that the clinically observed decreased erythrocyte survival and hepatic siderosis following portacaval shunts may result from hepatocellular damage produced by the diversion of the portal flow, which is superimposed on pre-existing liver damage.
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