Abstract
Summary
Norethynodrel caused enlargement of the corpora lutea with cellular hypertrophy and depletion of sudanophilic lipid and cholesterol. These changes appeared to be mediated by the hypophysis through the accelerated secretion of prolactin. On the other hand, ovarian interstitial tissue underwent involution with depletion of sudanophilic lipid and cholesterol. These effects were probably mediated by suppressed secretion of LH by the hypophysis.
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