Abstract
Summary
The acute effects of elevated renal venous pressure in development of reversible gross hematuria and oliguria were studied. Both isolated and intact dog kidney preparations were utilized. Results demonstrate that gross hematuria was produced as renal venous pressure was elevated, but not until a relatively high pressure was produced. Degree of hematuria increased as the pressure was further elevated, but disappeared rapidly following restoration to control pressure. The effect of renal venous pressure elevation on urine flow rate was one of progressive oliguria after a critical pressure was reached, and eventually anuria at the higher pressures. This effect was also reversible.
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