Abstract
Summary
The in vivo incorporation of C14-palmitate into liver and plasma triglycerides of rats fed a choline deficient diet has been studied. The results show that the accumulation of triglycerides occurring in the liver of these rats, unlike that which follows administration of carbon tetrachloride, ethionine and other hepatotoxic agents, is not due to an impairment of the movement of triglycerides from the liver to the plasma. Choline-deficiency fatty liver has thus a pathogenetic mechanism different from that of fatty livers due to toxic agents.
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