Abstract
Summary
Hypercholesteremia in rhesus monkeys was associated with defective utilization of glucose as evidenced by diminished glucose tolerance and increased plasma NEFA values. Hypercholesteremic rhesus monkeys excreted increased amounts of 17-ketosteroids and had diminished plasma insulin-like activity. The impaired carbohydrate metabolism of these animals was possibly due to combined effects of insulin insufficiency and hyperfunction of the adrenal cortex.
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