Abstract
Summary
Riboflavin excretion by renal tubular cells would seem to use the transport system for organic bases. This conclusion is based on the fact that tolazoline selectively inhibits riboflavin while not impairing PAH excretion. PAH excretion was even enhanced by tolazoline presumably as a result of diversion of more blood to the portal circulation. Benemid can inhibit transport of both PAH and riboflavin.
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