Abstract
Summary
Hydroxylamine when administered to immobilized cats in acute experiments in doses of 10-20 mg/kg markedly reduced duration and spread of electrically-induced afterdischarges while increasing cerebral levels of γ-aminobutyric acid. These findings increase the probability that a causal relationship may exist between metabolism of γ-aminobutyric acid in a particular cerebral area and its excitability.
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