Abstract
The frequent occurrence of degenerative joint disease (DJD) pathology and the high degree of functional prejudice associated with it, justify the search for experimental models with a view to a better understanding of this pathology and its treatment. The models most commonly used resort to a sudden mechanical degradation by shock (post-contusive osteoarthritis) or to a kinematic disruption of the joint through more or less extensive menisco-capsulo-ligamentary lesions or lesions induced by toxic substances.
Cartilaginous lesions met with in human pathology are often secondary to a morphological anomaly at the origin of a hyperpressure. Maquet (1977) has suggested the bringing forward of the anterior tibial tubercle (ATT) in order to reduce the retro-patellar pressure. The drawing back by hollowing of this ATT appeared to us a suitable model for experimental osteoarthritis by hyperpressure.
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