Foster-Kennedy syndrome was described in 1911 as an ophthalmologic manifestation of compression by a solid tumor in the frontal area with intracranial hypertension (ICHT). We describe a peculiar case of Foster-Kennedy syndrome associated with an arteriovenous malformation in which neither optic nerve compression nor ICHT was obvious. We discuss the different pathogenic mechanisms to explain this case, for which a chronic venous hypertension was the most probabile etiology.
NevilleRGGreenblattSHKollaritsCR. Foster Kennedy Syndrome and an optociliary vein in a patient with a falx meningioma. J Clin Neuro-ophthalmol. 1984; 4: 97–101.
2.
JarusGDFeldonSE. Clinical and computed tomographic findings in the Foster Kennedy Syndrome. Am J Ophthal. 1982; 93: 317–322.
Fogarty-MackPPile-SpellmanJHacein-BeyL. The effect of arteriovenous malformations on the distribution of intracerebral arterial pressures. Am J Neuroradiol. 1996; 17: 1443–1449.
