Singh BN, Vaughan Williams EM. A third class of antiarrhythmic action. Effects on atrial and ventricular intracellular potentials, and other pharmacological actions on cardiac muscle, of MJ1999 and AH 3474. Br J Pharmacol39:675, 1970
2.
Singh BN, Vaughan Williams EM. The effect of amiodarone, a new antianginal drug, on cardiac muscle. Br J Pharmacol39:657, 1970
3.
Charlier R., Deltour G., Baudine A. et al. Pharmacology of amiodarone, an antianginal drug with a new biological profile. Arzneimittelforschung18:1408, 1968
4.
Singh BN, Hauswirth O.Comparative mechanisms of action of antiarrhythmic drugs. Am Heart J87:367, 1974
5.
Singh BN, Vaughan Williams EM. Effects of altering potassium concentration on the action lidocaine and diphenylhydantoin on rabbit atrial and ventricular muscle. Circ Res29:286, 1971
6.
Singh BNThe coming of age of class III antiarrhythmic principle: retrospective and future trends. Am J Cardiol78(suppl 4A):17, 1996
7.
Mason JW and EsvemInvestigators. A comparison of seven antiarrhythmic drugs in patients with ventricular tachyarrhythmias . N Engl J Med329:452, 1993
8.
TheCASCADE Investigators. The Cascade Study: randomized antiarrhythmic drug therapy in survivors of cardiac arrest in Seattle. Am J Cardiol72:280, 1993
9.
Wever EF, Hauer Rnw, Van Capelle FJL, et al. Randomized study of implantable defibrillator as first-choice versus conventional strategy in post-infarct sudden death survivors. Circulation91:2195, 1995
10.
Singh BN, Ahmed R.Class III antiarrhythmic drugs. Current Opin Cardiol19:12, 1994
11.
Sicouri S., Moro S., Elizari MVd-Sotalol induces marked action potential prolongation and early afterdepolarizations in M but not epicardial or endocardial cells of the canine ventricle. J Cardiovasc Pharmacol Therapeut2:27, 1997
12.
Nakaya Y., Martin DK, Campbell TJThe novel class III antiarrhythmic agent MS-551 blocks the cardiac inward rectifier with greater potency than sotalol or E4031: possible relevance to reverse use dependence. J Cardiovasc Pharmacol Therapeut2:39, 1997
13.
Antzelevitch C.Editorial comment: the M cell. J Cardiovasc Pharmacol Therapeut2:73, 1997
14.
Lukas A.Electrophysiology of myocardial cells in the epicardial, myocardial and endocardial layers of the ventricle. J Cardiovasc Pharmacol Therapeut2:61, 1997
15.
Singh BNArrhythmia control by prolonging repolarization: the concept and its potential therapeutic impact. Eur Heart J14:14, 1993
16.
Waldo AL, Camm AJ, de Ruyter H., et al. Effect of d-sotalol on mortality in patients with left ventricular dysfunction after recent and remote myocardial infarction. Lancet343:7, 1996
17.
Roden DMIbutilide and the treatment of atrial arrhythmias. Circulation94:1499, 1996
18.
Julian DG, Jackson FS, Prescott RJ, Szekely P.Control trial of sotalol for one year after myocardial infarction. Lancet1:1142, 1982
19.
Groh WJ, Gibson KJ, McAulty JH, Maylie JGβ-adrenergic blocking property of dl-sotalol maintains class III efficacy in guinea pig ventricular muscle after isoproterenol. Circulation68:77, 1995
Sicouri S., Moro S., Siri LN et al. Chronic amiodarone delays ventricular repolarization and reduces transmural dispersion of repolarization in the canine heart. PACE1911:638(Abstract), 1996
22.
Singh BNAmiodarone and homogeneity of ventricular repolarization and refractoriness . J Cardiovasc Pharmacol Therapeut1:265, 1996